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dc.contributor.authorGorno-Tempini, Maria Luisa
dc.contributor.authorOgar, Jennifer M.
dc.contributor.authorBrambati, Simona Maria
dc.contributor.authorWang, P.
dc.contributor.authorJeong, Je Hoon
dc.contributor.authorRankin, Katherine P.
dc.contributor.authorDronkers, Nina
dc.contributor.authorMiller, Bruce L.
dc.date.accessioned2019-05-29T17:57:44Z
dc.date.availableNO_RESTRICTIONfr
dc.date.available2019-05-29T17:57:44Z
dc.date.issued2006
dc.identifier.urihttp://hdl.handle.net/1866/22081
dc.publisherAmerican Academy of Neurologyfr
dc.titleAnatomical correlates of early mutism in progressive nonfluent aphasiafr
dc.typeArticlefr
dc.contributor.affiliationUniversité de Montréal. Faculté des arts et des sciences. Département de psychologiefr
dc.identifier.doi10.1212/01.wnl.0000237038.55627.5b
dcterms.abstractPatients with progressive nonfluent aphasia (PNFA) can become mute early in the course of the disease. Voxel-based morphometry showed that PNFA is associated with left anterior insula and inferior frontal atrophy. In PNFA with early mutism, volume loss was more prominent in the pars opercularis and extended into the left basal ganglia. Damage to the network of brain regions involved in both coordination and execution of speech causes mutism in PNFA.fr
dcterms.isPartOfurn:ISSN:0028-3878fr
dcterms.isPartOfurn:ISSN:1526-632Xfr
dcterms.languageengfr
UdeM.ReferenceFournieParDeposantGorno-Tempini, M.L., J.M. Ogar, S.M. Brambati, P. Wang, J.H. Jeong, K.P. Rankin, N.F. Dronkers, and B.L. Miller, Anatomical correlates of early mutism in progressive nonfluent aphasia Neurology, 2006. 67(10): p. 1849-51.fr
UdeM.VersionRioxxVersion publiée / Version of Recordfr
oaire.citationTitleNeurology
oaire.citationVolume67
oaire.citationIssue10
oaire.citationStartPage1849
oaire.citationEndPage1851


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